The Toy Poodle has been bred down from the Standard Poodle through the Miniature Poodle. By the middle of the 1950s this variety has so progressed in numbers the the English Kennel Club granted it a separate resister.
The Toy Poodle is a very active, intelligent and elegant-looking dog that carries himself proudly. The coat is very profuse and can be clipped to suit different tastes. All solid colours are permitted.
IMPORTANT. A Toy Poodle with exaggerated features is the result of bad breeding. Credit; The Breed Club Connection.
As a Toy Poodle owner, you can consider yourself lucky. Your cuddly little breed is not only a generally healthy one, it has a life expectancy of as long as 18 years. Of course, like all purebred dogs, the Toy Poodle does have its share of inheritable health problems.
SKIN TUMORS; These tumors are a result of abnormal cell growth on a dog’s skin. They appear as lumps that don’t go away, unless surgically removed. Skin tumors can be malignant—which spread to other areas of the body—or benign. A biopsy is the only way to determine what kind of tumor your dog has. If a lump is benign, your veterinarian will likely tell you to do nothing. But if malignant, a skin tumor must be treated aggressively. It’s best to have any skin lumps checked by your veterinarian.
BLADDER STONES; Just like humans, dogs can suffer from bladder stones—and they can be very uncomfortable for your Toy Poodle. Bladder stones occur when there are high concentrations of minerals in the urine. Bladder infections can also contribute to the onset of bladder stones. If your Toy Poodle is urinating more than usual, is unable to urinate, or has blood in his urine, contact your veterinarian immediately. These are all signs that your pet may be suffering from bladder stones or another urinary tract problem.
CATARACTS; When a Toy Poodle suffers from cataracts, the lens of the eyeball begins to get cloudy. If your dog has cataracts, you may notice that the center of his eye is no longer clear, and has become white. You may also notice that your dog is having trouble seeing and is walking into the walls and furniture.
PATELLAR LUXATION is the dislocation (slipping) of the patella (kneecap). In dogs the patella is a small bone that shields the front of the stifle joint. This bone is held in place by ligaments. As the knee joint is moved, the patella slides in a grove in the femur. The kneecap may dislocate toward the inside (medial) or outside (lateral) of the leg. This condition may be the result of injury or congenital deformities (present at birth). Patellar luxation can affect either or both legs.
The most common occurrence of luxating patella is the medial presentation in small or miniature dog breeds. Shallow femoral groove, weak ligaments and malalignment of the tendons and muscles that straighten the joint are all conditions that will predispose a dog toward luxating patellae.
Indications of patellar luxation are; difficulty in straightening the knee, pain in the stifle, limping, or the tip of the hock points outward while the toes point inward.
Grade 1: Intermittent patellar luxation – occasional carrying of the affected limb. The patella can easily be manually luxated at full extension of the stifle, but returns to proper position when pressure is released.
Grade 2: Frequent patellar luxation – in some cases luxation is more or less permanent. The affected limb is sometimes carried, although the dog may walk with the stifle slightly flexed.
Grade 3: Permanent patellar luxation – even though the patella is luxated, many animals will walk with the limb in a semi-flexed position.
Grade 4: Permanent patellar luxation – the affected limb is either carried or the animal walks in a crouched position, with the limb partially flexed.
LEGG-CALVE-PERTHES results when the blood supply to the femoral head is interrupted resulting in a vascular necrosis, or the death of the bone cells. Followed by a period of revascularization, the femoral head is subject to remodeling and/or collapse creating an irregular fit in the acetabulum, or hip socket. This process of bone cell dying and chipping followed by new bone growth and remodeling of the femoral head and neck, lead to stiffness and pain. The net results are similar to those experienced by larger breeds with hip dysplasia.” No specific causes of LCPD are known, although it is believed to have a genetic mode of inheritance, and is not believed to be caused by trauma alone. Because there is a genetic component, it is recommended that dogs affected with LCPD not be used in breeding programs.
PROGRESSIVE RETINAL ATROPHY (PRA) refers to a group of diseases affecting the retina at the back of the eye. These diseases cause the retinal cells to become increasingly abnormal over time. In most cases, the eventual outcome is blindness. Some form of PRA has been recognized in over 100 dog breeds, including Toy and Miniature Poodles.
PRA is inherited, meaning the disease genes that cause PRA are passed from generation to generation. In Toy and Miniature Poodles, one specific type of inherited PRA predominates, although at least one more type is present at a low frequency in the breed. This predominant form is the progressive rod-cone degeneration (prcd) form of PRA. Rod cells in the retina slowly lose function, with diminished vision in dim light and diminished field of vision. Subsequently, retinal cone cells lose function, resulting in diminished vision in daylight and eventual total blindness. The age of onset and the rate of disease progression are variable among different breeds, within the same breed and within the same litter. Â In general for Toys and Miniatures, diagnosis of prcd-PRA is made around 3 years of age, based on an eye exam by a veterinary ophthalmologist. Some prcd-PRA affected dogs retain some useful vision throughout life, while others progress to blindness in mid-life. Unfortunately, there is no treatment or cure for PRA.
ADDISON’S DISEASE is also extremely common in Poodles. In dogs with this condition, the adrenal glands don’t produce enough of the hormone cortisol. The dogs become lethargic, depressed and intolerant of stress, and they may have digestive problems.
Poodles are also at high risk of the opposite of Addison’s, the condition called Cushing’s Syndrome. These dogs’ adrenal glands produce too much cortisol. Symptoms include weight gain, panting, excessive thirst and hunger, bladder infections and urinating in the house even though the dog was previously house-trained Cushing’s is usually managed with lifelong medication, but sometimes requires very expensive and difficult surgery to correct.
Another hormonal problem common in Poodles is thyroid disease. Symptoms include weight gain, hair loss, lack of resistance to disease, excessive hunger and seeking out warmth. Thyroid hormone supplements can be given, but, like Addison’s, the condition can be hard to manage medically.
CHRONIC ACTIVE HEPATITIS is a liver disease where there is inflammation of the liver and death of liver tissue present. Dogs that are affected with this disease develop a slow, progressive liver failure. Researchers have found in some breeds a familial predisposition to the disease. In Bedlington Terriers, the disease has been found to be the result of an autosomal recessive gene, and there is a marker test from Vetgen to test for it. In Dobermans, the disease seems to affect more females than males.
Symptoms of the illness usually don’t appear in the dogs’ early years, not until there is significant damage to the liver. Usually CAH appears around 5-7 years of age. Some of the early signs of CAH are loss of appetite, vomiting, diarrhea, vomiting yellowish bile, weight loss, depression, increased water intake, increased urination, and sluggishness. As the disease gets worse, jaundice may appear (the whites of the dogs eyes will appear yellowish), and clotting problems may occur. Fluids can build up in the abdominal area, so that a dog may look like it’s in whelp. Behavioral changes may occur, such as the dog might stand and stare at the wall, or in a corner, or just stand and be confused. All of this is due to toxins that have built up in the body that used to be metabolized by the liver.